Gastrointestinal Pathology: An Atlas and Text by Cecilia M. Fenoglio-Preiser, Amy E. Noffsinger, Grant N.

By Cecilia M. Fenoglio-Preiser, Amy E. Noffsinger, Grant N. Stemmermann, Patrick E. Lantz, Peter G. Isaacson

Thoroughly up-to-date for its 3rd version, this full-color text/atlas is a entire reference on gastrointestinal pathology in adults and pediatric sufferers. The e-book integrates pathologic analyses of specimens with anatomic, medical, radiologic, pathophysiologic, and microbiologic details to provide readers a transparent knowing of every illness strategy. the phenomenal collection of over 3,000 illustrations comprises gross and microscopic images, electron micrographs, radiographs, and line drawings.

Chapters hide non-neoplastic and neoplastic lesions of every anatomic part and problems affecting a number of anatomic websites. This version contains many new illustrations, major updates on mesenchymal tumors, motility issues, and inflammatory bowel affliction, and new details on molecular biologic adjustments in a variety of disorders.

A significant other web site will provide the totally searchable textual content and a picture financial institution.

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By Cecilia M. Fenoglio-Preiser, Amy E. Noffsinger, Grant N. Stemmermann, Patrick E. Lantz, Peter G. Isaacson

Thoroughly up-to-date for its 3rd version, this full-color text/atlas is a entire reference on gastrointestinal pathology in adults and pediatric sufferers. The e-book integrates pathologic analyses of specimens with anatomic, medical, radiologic, pathophysiologic, and microbiologic details to provide readers a transparent knowing of every illness strategy. the phenomenal collection of over 3,000 illustrations comprises gross and microscopic images, electron micrographs, radiographs, and line drawings.

Chapters hide non-neoplastic and neoplastic lesions of every anatomic part and problems affecting a number of anatomic websites. This version contains many new illustrations, major updates on mesenchymal tumors, motility issues, and inflammatory bowel affliction, and new details on molecular biologic adjustments in a variety of disorders.

A significant other web site will provide the totally searchable textual content and a picture financial institution.

Show description

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Additional info for Gastrointestinal Pathology: An Atlas and Text

Sample text

Doublecontrast esophagram demonstrates numerous irregular thin outpouchings from the esophageal lumen, some of which have flask-shaped bases. These represent ectatic submucosal glands. A B FIG. 30. Diffuse esophageal intramural pseudodiverticulosis. A: Low-power magnification demonstrating the presence of a cystically dilated duct, which passes between lobules of the submucosal glands. Some of the ducts have also undergone squamous metaplasia. B: Higher magnification of one of the ducts showing the presence of inspissated secretions and flattened cuboidal lining epithelium.

The lamina propria rests on a two-layer, relatively thick muscularis mucosae. qxd 09/13/2007 8:14 AM Page 16 Aptara Inc. 16 Gastrointestinal Pathology: An Atlas and Text Normal Histology at the Gastroesophageal Junction FIG. 8. Lymph nodes draining the esophagus. See text for further description. integrity and cell maturation. The epidermal growth factor receptor (EGFR) possesses tyrosine kinase activity (16) and binds EGF. This may make the esophageal mucosa vulnerable to the anti-EGFR therapies used to treat multiple types of cancer.

B: Higher magnification showing the gastric epithelium. smaller lesions, as compared to larger ones, suggesting that the lymphocytes play a role in lesional regression. An intense inflammatory reaction may surround the lesion, especially following peptic ulceration. tissues. Inflammation and necrosis follow. Heterotopic pancreas differs from pancreatic metaplasia, which is discussed in a later section. The latter is usually a focal change consisting only of pancreatic acini that blend into cardiac Heterotopic Gastric Mucosa away from Inlet Patches Ectopic gastric mucosa also occurs in the mid- or distal esophagus, usually in congenital malformations, including duplications or diverticula.

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