By Jacqueline Jumpsen
'This book's target is to supply a centred assessment (morphological, biochemical and useful) of mind improvement and to exemplify the position of lipids within the very important developmental occasions and the ideas which are almost certainly altered by means of physiological alterations in mind lipid composition. person chapters contain: mind progress and vulnerability; mind lipids; Lipids and crucial fatty acids in mind improvement; crucial fatty acid synthesis and shipping to the mind; impression of nutrition on fatty acid composition in mind; Glycolipids and gangliosides; Neurotransmitters; and Relevance, reflections and destiny directions.'
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Extra info for Brain development : relationship to dietary lipid and lipid metabolism
Example text
1982; Sinclair, 1975).
Analyses of whole-body fat content (Widdowson, 1968, 1979) indicate that preterm infants, with an appropriate weight for gestational age of 1300 g at birth, have a total body fat content of about 30 gm compared with the term infant of 3500 g with a total body fat content of 340 g. Clandinin et al. (1981a) estimated that the adipose tissue of an infant born at 30 weeks gestation, weighing 1300 g, would contain 460 g of n-6 essential fatty acids and 66 gm n-3. Since approximately 2783 mg of n-6 fatty acids and 387 mg n-3 fatty acids accrue in adipose tissue each week in utero (Values = Means ± 2SD), birth after only a few more weeks of intrauterine development would dramatically increase the potential reserve of fatty acids in adipose tissue for both total fatty acids, used for energy production, and for essential fatty acids, used for synthesis of structural tissues.
The essential nature of linoleic and α-linolenic acids in the diet produces a relationship between diet, membrane structural lipid and function through the synthesis of new fatty acids by de novo mechanisms, and the incorporation of acyl groups into membrane lipids from phospholipid acyl group turnover. The levels of 18:2n-6 and 18:3n-3 present in the diet can affect the resulting levels of their respective homologues. , 1981a). A deficiency for both n-6 and n-3 fatty acids has been shown to cause physical and biochemical changes.